Login
 

Bronchitis

Bronchitis refers to the inflammation of the large and medium bronchi. Acute bronchitis is typically the result of an acute viral URI of the pharynx, throat, and bronchial tree, sometimes with secondary bacterial infection. Fever, lymphadenopathy, myalgia and other symptoms of a URI are typically present (see Herbal Immunity: Nonspecific resistance, Immunity and Botanical medicine<). Viruses that cause acute bronchitis include adenovirus, coronavirus, influenza A and B viruses, parainfluenza virus, respiratory syncytial virus, coxsackievirus, rhinovirus, and the viruses that cause rubella and measles. Bacterial causes include Mycoplasma pneumoniae, Bordetella pertussis and Chlamydia pneumoniae. Acute bronchitis may also be caused by acute exposure to various dusts, fumes and smoke. Acute bronchitis is usually a self-limiting condition in most patients, with maximal symptoms occurring within three to five days after the onset of the condition, resolving over a two-week period. Complications usually only occur in patients with an underlying respiratory illness, including bronchiolitis and bronchopneumonia. (Berkow 1992; Govan 1991, 299)

Acute bronchitis is characterized by mucosal inflammation and the abundant production of sputum that is often mucopurulent. As the sputum accumulates in the bronchi it initiates the cough reflex, which along with the ciliated epithelia, allows the sputum to be cleared from the air passages. In some cases dyspnea results from edema and spasm of the bronchial walls. Upon auscultation the breath sounds may exhibit occasional crackling, scattered ronchi, and wheezing after coughing. (Berkow 1992; Govan 1991, 299)

Along with emphysema and asthma, chronic bronchitis is a chronic pulmonary obstructive disorder (CPOD) that is defined as a chronic, productive cough experienced for more than a two-year period. The primary pathological features are characterized by an increase in goblet and mucus cells with a commensurate loss of serous glands and ciliated epithlium, resulting in a thick, viscous sputum that is difficult to expectorate. With repeated inflammation there is fibrosis and a thickening of the bronchial wall, which further impairs airflow. In progressed conditions hypertrophy of the right heart ventricle (cor pulmonae) can occur. In most cases the patient is a smoker, although environmental pollution is another important factor and is probably an increasing trend, especially in highly congested urban areas. Nutrient deficiencies such as vitamin A, essential fatty acids and accessory antioxidants also facilitate the condition. (Berkow 1992, 658-661; Rubin and Farber 1990, 328; Govan 1991, 300-301)

Medical treatment

In acute bronchitis bed rest and hydration are the usual therapies, along with antipyretics such as ASA and acetaminophen. Antitussives used to inhibit or suppress the cough reflex do so by depressing the medullary cough center, and include drugs such as chlophedianol, levopropoxyphene, dextromethorphan, and codeine. Expectorants are also used to help expel the congested sputurm from the respiratory tract by decreasing its viscosity, and include iodides (side-effects include acne, coryza, erythema of face and chest, painful swelling of the salivary glands, and hypothyroidism with long term use), syrup of ipecac (nausea and vomiting), guaifenesin (generally well-tolerated), ammonium chloride, terpin hydrate, and even creosote. Demulcents are often used as an adjunct to antitussive preparations including acacia, glycerin, honey, sugar syrup (a questionable antitussive) and sometimes an extract of Prunus virginiana bark. Antibiotics are the mainstay of treatment in purulent acute bronchitis, including tetracycline, erythromycin, amoxicillin or ampicillin: hopefully the choice is based on a cytological analysis of a sample of the sputum.

The treatment of chronic bronchitis is directed towards the removal of the cause, which may include regular vaccination if chronic bronchitis is a common sequela to a URI, and smoking cessation. Patients may undergo allergy testing to determine the presence of potential allergens, and may be recommended desensitization therapy in weekly injections. Symptomatic therapy consists of bronchodilators such as ?2-agonists (e.g. metaproterenol, albuterol, terbutaline, and pirbuterol) and anticholinergics (e.g. ipratropium). In come cases corticosteroids may be prescribed, either orally or topically, but there is little evidence of their benefit. Antibiotics are also sometimes used in acute exacerbations of chronic bronchitis, including increased cough, catarrh, dyspnea, and fever. In more severe forms of chronic bronchitis oxygen therapy may be administered.

Holistic treatment

In Ayurvedic medicine kasa (cough) is caused by the inhalation of dust and noxious fumes, excessive exercise, foods that are dry, and the suppression of natural urges. There are five variants of kasa, including Vataja (dry cough), Pittaja (heat and inflammation), Kaphaja (swelling and mucus), Kshataja (caused by injury) and Kashaya (caused by asthenia and wasting, i.e. tuberculosis). In Chinese medicine bronchitis is differentiated based on the pathological factors, including the Retention of Phlegm with Extrinsic Cold (coryza with whitish sputum, floating pulse), Stagnation of Phlegm-Heat (thick yellow sputum, rapid pulse), and a Lung and Spleen Qi Deficiency (weak cough, SOB, thin and deep pulse). From a Western herbal perspective the causes of acute and chronic bronchitis are identical to that of modern medicine, although underlying factors are taken into consideration, include diet and nutrition, immune status and the efficiency of eliminative organs.

1. Ease cough.

  • Demulcents and vulneraries, used in acute inflammation and dryness, and not in profound catarrh, e.g. Licorice (Glycyrrhiza glabra), St John’s Wort (Hypericum perforatum), Comfrey (Symphytum officinale), Plantain (Plantago spp), Marshmallow (Althaea officinalis), Chickweed (Stellaria media), Mai Men Dong (Ophiopogon japonicus), Shi Di Huang (Rehmannia glutinosa), Shi Hu (Dendrobium nobile)
  • Stimulating expectorants, used in highly congestive conditions with a thick profuse catarrh e.g. Heartsease (Viola tricolor), Squill (Urginea maritima), Stillingia (Stillingia sylvatica), Primrose (Primula vulgaris), Daisy (Bellis perennis), Senega (Polygala senega), Euphorbia (Euphorbia hirta), Guggulu (Commiphora mukul), Cottonwood (Populus candicans), Grindelia (Grindelia camporum).
  • Respiratory antispasmodics, used in bronchial constriction, and spasmodic (whooping) cough, e.g. Jimsonweed (Datura stramonium), Ephedra (Ephedra sinica), Visnag (Ammi visnaga), Lobelia (Lobelia inflata), Wild Cherry bark (Prunus serotina), Mullein (Verbascum Thapsus), Elecampane (Inula helenium), Cottonwood (Populus candicans), Thyme (Thymus vulgaris), Sundew (Drosera rotundifolia), Grindelia (Grindelia camporum), Wild Lettuce (Lactuca virosa), Bloodroot (Sanguinaria canadensis),
  • Mucolytic expectorants, to decrease viscosity of mucus secretions, e.g. Ginger (Zingiber officinale), Cardamom (Elettaria cardamomum), Chinese Cinnamon bark (Cinnamomum cassia), Anise (Pimpinella anisum), Cayenne (Capsicum minimum), Prickly Ash (Zanthoxylum clavaherculis), Garlic (Allium sativum) and Dang Gui (Angelica sinensis).
  • Astringing expectorants, to dry up excessive mucus secretions, e.g. Bayberry (Myrica cerifera), Eyebright (Euphrasia officinalis), Canada Balsam (Abies balsamera), Goldenrod (Solidago virgaurea), Mullein (Verbascum thapsus), Goldenseal (Hydrastis canadensis)

2. Remove causes.

  • quit smoking (tobacco, cannabis)
  • limit exposure to environmental toxins, especially in air-conditioned indoor environments where viral, bacterial and fungal pathogens are continuously re-circulated. Personal air filters in such environments are recommended.
  • address the issue of dietary intolerances and food allergens by implementing a elimination-challenge diet
  •  Generally speaking heavy and sticky foods such as dairy, sweets and flour products should be avoided.
  • assess patient for air-borne allergens (see Asthma)

3. Treat infection and reestablish the body’s ecology.

  • Antivirals: St John’s Wort (Hypericum perforatum), Nu Zhen Zi (Ligusticum lucidum), Biscuit root (Lomatium spp), Bhunimba (Andrographis paniculata), Guduchi (Tinospora cordifolia), Ban Lan Gen (Isatis tinctoria)
  • Antibacterials: Purple Coneflower (Echinacea angustifolia), Wild Indigo (Baptisia tinctoria), Goldenseal (Hydrastis canadensis), Garlic (Allium sativum), Guggulu (Commiphora mukul), Nimba (Azadirachta indica), Bhunimba (Andrographis paniculata), Katuka (Picrorrhiza kurroa), Guduchi (Tinospora cordifolia), Haridra (Curcuma longa), Huang Lian (Coptis chinense), Lian Qiao (Forsythia suspens), Jin Yin Hua (Lonicera japonica), Ban Lan Gen (Isatis tinctoria), Huang Qin (Scutellaria baicalensis)
  • Antifungals: Garlic (Allium sativum), Sweet Annie (Artemisia annua), Pau D’Arco (Tabebiua spp.), Barberry (Berberis vulgaris), Purple Coneflower (Echinacea angustifolia), Spilanthes (Spilanthes acmella), Nimba (Azadirachta indica), Huang Lian (Coptis chinense), Tulasi (Ocimum sanctum), Bhringaraj (Eclipta alba), Haritaki (Terminalia chebula), Hingu (Ferula foetida) and Bibhitaki (Terminalia bellerica)
  • Probiotics: e.g. live culture foods, lactobacilli, bifidobacterium
  • Prebiotics: e.g. fructo-oligosaccharides, inulin (e.g. found in Inula and Taraxacum root)

4. Support immune function.

  • Lymphagogues as supportive, and specifically with lymphadenopathy, e.g. Purple Coneflower (Echinacea angustifolia), Redroot (Ceanothus americanus), Poke Root (Phytolacca americana), Cedar (Thuja spp), Cleavers (Galium aparine), Red Clover (Trifolium pratense)
  • Immunomodulants in chronic or recurring conditions, e.g. Purple Coneflower (Echinacea angustifolia), Pau D’Arco (Tabebiua spp.), Reishi (Ganoderma lucidum), Huang qi (Astragalus membranaceus), Amalaki (Emblica officinalis), Wu Wei Zi (Schizandra chinense)
  • Immunosupportive nutrients, including vitamins A (25,000 IU daily), B complex (50 mg daily), C (to bowel tolerance) and E (800 IU daily), as well as zinc (50 mg daily)

5. Clear the body of toxins. Of particular in importance where chronic bronchitis represents the cumulative effects of congestion and stasis in other eliminative systems.

  • Cholagogues and hepatotrophorestoratives to enhance liver detoxification with cholagogues and supportive nutrients e.g. Barberry (Berberis vulgaris), Boldo (Peumus boldus), Milk Thistle (Silybum marianum), Haritaki (Terminalia chebula), Haridra (Curcuma longa), Guduchi (Tinospora cordifolia). Bhumyamalaki (Phyllanthus amarus), Katuka (Picrorrhiza kurroa), Huang Qin (Scutellaria baicalensis),
  • Diuretics: Priest and Priest state that in addition to liver detoxification the kidneys maintain a special relationship with the lungs, and thus an increase in respiratory catarrh can indicate a relative insufficiency of renal function (1982, 10-11), e.g. Celery seed (Apium graveolens), Cleavers (Galium aparine), Nettles (Urtica dioica), Goldenrod (Solidago virgaurea), Horsetail (Equisetum arvense)
  • Lymphagogues: Poke Root (Phytolacca americana), Cedar (Thuja spp), Cleavers (Galium aparine), Redroot (Ceanothus americanus)
  • Hydration and heat: showers, baths, steam baths, sweating under blankets; drinking 2 liters of water daily

6. Hydrate nasal and sinus mucosa.

  • Neti, with isotonic water, twice daily
  • Humidification, especially at night, with essential oils (e.g. Spruce, Eucaplytus, Rosemary, Cedar, Pine, etc.)
  • Nasya, 2-3 gtt of sesame oil instilled and inhaled into each nostril, once daily on an empty stomach in the morning
  • Demulcents: Marshmallow (Althaea officinalis), Slippery Elm (Ulmus fulva), Licorice (Glycyrrhiza glabra), Kumari juice (Aloe vera)

7. Dietary changes.

  • Mucus-producing foods must be eliminated, including dairy, flour, and sugar.
  • Animal proteins and fats should be reduced in favor of lightly cooked vegetables and boiled whole grains for the duration of the illness.
◄ Bilious Dyspepsia   Cancer ►